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Recruitment of the p97 ATPase and ubiquitin ligases to the site of retrotranslocation at the endoplasmic reticulum membrane

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Furthermore, GHK-Cu's potential mechanism might involve the facilitation of copper uptake by cells, which might then be utilized by intracellular copper-dependent enzymes or directly activate transcription factors

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We propose that all considered effects of GPX4 on the risk of CS can be mediated by three following mechanisms