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These effects are strongly supported by molecular findings, such as the upregulation of anabolic pathways (mTORC1) and the downregulation of catabolic and autophagic signaling, as well as reduced tumor progression

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Under persistent attack by periodontopathic bacteria, the regulatory direction of autophagy undergoes a fundamental shift: ferritinophagy is activated to release iron, whereas mitophagy is suppressed to accumulate ROS

6) and tumour growth (Fig