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Moreover, the LN microenvironment accelerates epigenetic (60) and metabolic reprogramming of cancer cells (61, 62) that promote resistance to ferroptosis, enhancing survival and metastasis through the blood (62)

Mapping the functional interactions at the tumor-immune checkpoint interface

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The higher GSH concentration in the cytoplasm facilitates the disruption of disulfide bonds within LNP structures incorporating the CR8C peptide, enabling improved gene therapy [165]