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Although the residues such as W125, L128, C132, and M138, which form this pocket, were previously implicated in the BubR1-KNL1 KI2 interaction, their contribution to BubR1 kinetochore appears limited

In a study, Fuchs et al

This review delineates the multifaceted mechanisms underlying metabolic dysfunction-associated steatotic liver disease (MASLD), with an emphasis on systemic metabolic remodeling, mitochondrial protection, and intracellular calcium restoration

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