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Elucidating CYP2D6-driven metabolism and hepatotoxic bioactivation of metoprolol in plateable human and animal hepatocytes Original scientific paper DOI: Keywords: Metabolite, cytotoxicity, recombinant, low-clearance, glutathione, CYP2D6 Abstract Background and purpose: As a classic -blocker with low systemic clearance, metoprolol has been linked to rare but clinically significant hepatotoxicity, yet its hepatic metabolic fate remains poorly characterized

Microbial flora of the mouse ileum mucous layer and epithelial surface

A small number of NSCs remain in adults even if most of them are quiescent (recently reviewed in Zhao and Moore, 2018)

For instance, ROS-mediated damage impairs the function of immune cells, reducing their ability to effectively combat infections while simultaneously promoting chronic inflammation ( 3.2 Immune cell dysfunction and the gut-kidney axis in CKD Immune cell dysfunction is a key driver of CKD progression (Figure 2), involving abnormalities in adaptive and innate immune cells, including T cells, B cells, and monocytes/macrophages

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