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[DOI] [PMC free article] [PubMed] [Google Scholar] 70.Odenwald MA, Turner JR

This happened most probably by efficiently increasing a subcellular pool of GSH available for this aim, as demonstrated by the substantially higher consumption of GSH and production of its oxidized form, GSSG, in NACET-pretreated cells subjected to oxidative insult in comparison with untreated or NAC-pretreated cells