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Description

The mechanism is hypothesized to be similar to the intracellular process that leads to neuronal damage in Alzheimers disease

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Monitoring Regular check-ups are essential to track cardiac performance and adjust dosing

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Differential regulation of resolution in inflammation induced by amyloid-beta42 and lipopolysaccharides in human microglia

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NG2+ CNS glial progenitors remain committed to the oligodendrocyte lineage in postnatal life and following neurodegeneration

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Inflammation markers, such as leukocytes, are recruited from the damage sites and then participate in the increased uptake of oxygen for the release of ROS and thus its accumulation

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