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Magnesium deficiency in the brain leads to platelet aggregation and the release of glutamate, resulting in the formation of 5-hydroxytryptamine (5-HT)

population, with approximately 50% of SOD1-ALS patients carrying the A4V mutation

Future directions Available research on AOX therapy is surrounded by several limitations and biases, including small sample sizes, heterogenous patient populations and inclusion criteria, poor study designs, and inconsistent reporting of clinical outcomes

Such fluctuations in the SM profile could be due to the type of helminth species used for infection or duration of infection and animal models used