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Tragically, the public debate has not yet moved toward its postnatal use, for which relative and absolute risks are greater and evidence of neurodevelopmental injury is conclusive [2]

By genetically deleting the disulfide reductase pathways from mouse liver, we uncovered an inducible, ubiquitous, cell-autonomous reductase-independent backup pathway for the generation of cysteine, driven by cystine CS bond cleavage

TC is even more promiscuous in its binding than IF and so it will transport both the intact and degraded B12 into the cell

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