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In PCOS granulosa cells, despite the absence of evidence directly substantiating Cer-mediated pro-senescence effects, this bioactive sphingolipid has been demonstrated to elicit oxidative stress [12], mitochondrial dysfunction [22], and DNA damage [23], all of which are critical drivers of cellular senescence [24]

These patients need to understand that with time, treated veins may recur or new veins may develop

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they work by binding bile acids in the intestine so theyre carried out in stool instead of being reabsorbed.48 Some aluminum-based antacids (like aluminum hydroxide) can bind bile acids, too.49 Because these products trap bile acids in the gut, they can trap TUDCA as well reducing its absorption and likely blunting any effect

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