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In ischemiareperfusion and inflammatory AKI models, paeoniflorin treatment suppressed renal TLR4 expression, inhibited NF-B activation, and significantly reduced pro-inflammatory cytokine production, including tumor necrosis factor- (TNF-) and interleukin-1 (IL-1), thereby attenuating inflammatory cell infiltration and microvascular dysfunction in injured kidneys (Zhang et al

While inhibiting NOS may seem to be an obvious therapy, this is complicated by the fact that nitric oxide is integral for physiological function, particularly for the vascular system

Importantly, youre getting clinical trial level doses in each capsule

An adduct between TRi-1 was detected by TLC and LCMS analysis (Supplementary Fig

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