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showed that genetic polymorphisms in vitamin D metabolism-related enzymes were associated with the risk and severity of ASD 31 , and a meta-analysis suggested that vitamin D receptor rs731236 and rs7975232 were significantly associated with ASD 32

These changes can be reversed by Fer-1, indicating that iron overload induced ferroptosis in HH mice, and suggesting targeting ferroptosis could be a promising strategy for treating hemochromatosis-related injury

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Based on studies of plants deficient in enzymes such as ascorbate peroxidase (APX) and catalase (CAT), accumulation of GSSG is quite closely related to the intracellular availability of H 2 O 2 (Rizhsky et al., 2002