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Comment on recent modeling studies of astrocyteneuron metabolic interactions

Despite the progress made in comprehending the mechanisms of UA-induced inflammation, there remains an ambiguity concerning how UA regulates immune cells involved in the inflammatory response in HUA and its complications

Transcriptomic analyses in middle cerebral artery occlusion (MCAO) models indicate that HBOT modulates inflammatory signaling, bloodbrain barrier integrity, neural repair pathways, and RNA epigenetic modifications, including reduced m6A methylation levels (49)