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For instance, HDAC1, HDAC2, and HDAC3, which belong to class I HDACs, have been demonstrated to mediate the deacetylation of mitogen-activated protein (MAP) kinase phosphatase 1 (MKP1), resulting in elevation of MAP kinase (MAPK) and suppression of innate immune signaling [62]

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Jun 7 2017;18(6)doi:10.3390/ijms18061221

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While the functional relevance of suboptimal riboflavin status in otherwise healthy individuals is not clear, in vitro studies have shown increased protein carbonylation, DNA strand breaks, and cellular stress and apoptosis with riboflavin deficiency associated with little to no glutathione reductase activity (211, 340)