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The role of the gut as a mediator is unequivocally demonstrated in models of chronic apical periodontitis (CAP), where P.g -induced CAP alters gut microbiota (increasing Lachnospiraceae , Ruminococcaceae ) and increases TMAO, thereby exacerbating aortic plaque formation

In addition to MTHFR gene mutations, several other things can contribute to high homocysteine levels, including: Kidney disease: Kidney disease can damage the kidneys' ability to filter homocysteine from the blood

Extranuclear localization of SIRT1 and PGC-1: an insight into possible roles in diseases associated with mitochondrial dysfunction

To understand the effects of estradiol on such a complicated network, mathematical computation is required

The increase in Th2-associated cytokines can perpetuate systemic inflammation and exacerbate hallmark symptoms such as fatigue and musculoskeletal pain [57]