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doi: 10.1093/jnci/djg061 147 FanCChenJWangYWongYSZhangYZhengWet al

In preclinical models of cerebral ischemia, GLPs significantly diminish cerebral infarct volume and attenuate neuronal cell death by modulating the mitochondrial apoptotic pathwayspecifically, by upregulating the anti-apoptotic protein Bcl-2, downregulating the pro-apoptotic factor Bax, and suppressing the activation of executioner caspases (caspase) (Liu X