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In HCC, NRF2 overexpression drives a multifaceted program that includes metabolic reprogramming, immune evasion, and chemoresistance

l-glutathione (GSH) chemically is (2 S )-2-amino-5-[[(2 R )-1-(carboxymethylamino)-1-oxo-3-sulfanylpropan-2-yl] amino]-5-oxopentanoic acid (Fig

(2021) clearly indicated that hydrogen-saturated saline mediates neuroprotection through autophagy via the PI3K/AKT/mTOR pathway during the early and medium stages of rotenone-induced PD in rats