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This separation is important: semaglutide does not block glutathione synthesis, and glutathione does not impair GLP-1 signaling

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Targeting metabolic vulnerabilities in specific macrophage subsets, such as limiting ferroportin-mediated iron export in M2 TAMs or enhancing ACSL4-dependent lipid peroxidation in inflammatory TAMs, may shift the TME toward a ferroptosis-permissive state