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SECONDARY HYPERPARATHYROIDISM IN CKD CLINICAL FEATURES SECONDARY HYPERPARATHYROIDISM IN CKD -- DIAGNOSIS SECONDARY HYPERPARATHYROIDISM IN CKD TREATMENT ACKNOWLEDGEMENTS REFERENCES [Changes in mineral metabolism in stage 3, 4, and 5 chronic kidney disease (not on dialysis)].[Nefrologia

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For the first time, monophenolase activity of jr PPO1 towards L -tyrosine was blocked in two double mutants (Asn240Lys/Leu244Arg and Asn240Thr/Leu244Arg) while its diphenolase activity was partially preserved, thereby converting jr PPO1 into a CO

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Liver-metastatic breast cancer cells display a distinct metabolic reprogramming characterized by accumulation of glucose-derived lactate and reduction in the TCA cycle and OXPHOS (138)

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