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This biochemical mechanism supports the observed neuroprotective effects, as NAC enhances intracellular glutathione production, thereby reinforcing the cellular redox system, promoting detoxification of HNE, and ultimately protecting neurons from oxidative damage

In the MTX group, severe vacuolar degeneration and pyknotic nucleus in hepatocytes, increased number of activated Kupffer cells, inflammatory cell infiltration and marked sinusoidal dilatation and congestion were prominent features of the morphologic damage (Fig

Five randomly selected fields were analyzed per section