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In a physiological state, PERK, IRE1, and ATF6 are non-covalently bonded to glucose-regulated protein 78 (GRP78)

Lastly, ferroptosis can trigger systemic inflammatory response, activate multiple pro-inflammatory signaling, induce cardiomyocyte hypertrophy, fibrosis and death, and eventually lead to chronic adverse ventricular remodeling (Shirazi et al., 2017

doi: 10.1016/j.mam.2008.08.006 8