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Importantly, FDX1 is considered a central modulator in cuproptosis due to the fact that its absence leads to a total loss of protein lipoylation, a substantial decrease in cellular respiration, accumulation of pyruvate and -ketoglutarate, reduction of succinate, and stabilization of Fe-S cluster proteins

Inhibition of NADPH oxidase promotes alternative and anti-inflammatory microglial activation during neuroinflammation

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Consistent with these findings, GSH-depleted mice showed significantly decreased weights of liver and epididymal (visceral) white adipose tissue (eWAT) (Figure 2, D and E), while weights of inguinal (subcutaneous) white adipose tissue (iWAT), brown adipose tissue (BAT), or skeletal muscle remained unchanged (Supplemental Figure 2, FH)

Renal cancer cells have been reported as highly dependent on the GSH pathway for ROS detoxification, including lipid peroxides, and targeting components of this pathway is an effective strategy for the treatment of this disease