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Previous studies have confirmed the above view that in the post-arthroplasty tissues of patients with RA, RNAKL is mainly expressed at the endothelial-bone interface and subchondral bone erosion sites, the site of contact between the pannus and cartilage (236)

One possibility is DsbC and it has been suggested that reduced glutathione can react with the isomerase, especially by providing reductive power when DsbD is missing (Pittman et al., 2005

It challenges the established notion of heightened immunogenic potential post late-stage cell demise, traditionally attributed to a surge in inflammatory mediators upon cell rupture

doi: 10.1016/j.colsurfb.2023.113487 118