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Stressed and damaged cells, as a result of altered metabolic and physiological states, can produce ROS as a byproduct

10.1111/ijcp.14724 327 MirzaeiR.BouzariB.Hosseini-FardS

Cancer Risk A review and discussion stated: ROS production is significantly increased in cancer cells because of mitochondrial dysfunction, altered metabolism, and frequent genetic mutations, resulting in an accumulation of large amounts of oxidized protein, DNA, and lipids as an adaptive response, cancer cells harbor elevated levels of ROS-scavenging molecules

It works by inhibiting the tyrosinase enzyme to slow melanin production while supporting cellular repair and rejuvenation

Tracking genetic variants in the biomedical literature using LitVar 2.0