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Once liberated, AA acts as the primary substrate for the COX and lipoxygenase (LOX) pathways, leading to the synthesis of eicosanoids such as PGE2 and leukotrienes

It acts as a mitochondrial protective agent, stabilizing mitochondrial structure, enhancing oxidative phosphorylation, reducing production of reactive oxygen species (ROS), and mitigating cellular damage in diverse models of mitochondrial dysfunction

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Using this bacterium as a model, we identified a role for GR in intricately managing the balance of GSH and GSSG, which is essential for neutralizing ROS and protecting against oxidative cellular damage