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In summary, SLC7A5 overexpression was able to reverse the protective effects of YTHDF1 knockdown, revealing the critical role of SLC7A5 in the YTHDF1-mediated cytoprotective mechanism

This makes melanocytes relatively vulnerable to oxidative stress [23]

Doris Wilflingseder initiated the MUI AnimalFree Research Cluster at the Medical University of Innsbruck (MUI)

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