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Disturbances in the endoplasmic reticulum can be triggered by increased oxidative stress, deposition of saturated fatty acids, hypoxia, or other stimuli, which in turn cause the accumulation of unfolded proteins in the endoplasmic reticulum lumen that is filled with proteins, antioxidants, or essential metals

Additionally, exposure of erythrocytes to heavy metals, pollutants, and xenobiotics triggers eryptosis through various mechanisms of cell death

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This kind of protection-damage dynamic transformation can well explain the phenomenon of the bilirubin paradox in PD (Moccia et al., 2015

(1998) ADP-ribosylation factor 1 (ARF1) regulates recruitment of the AP-3 adaptor complex to membranes