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doi: 10.3389/fmicb.2015.00584

While further research is essential, the comprehensive effects of KD on PD-related pathophysiology position it as a promising non-pharmacological strategy

[161] reported that glutathione could reverse increased IL-17a and LC3-II levels and decrease Foxp3 and P62 levels in AR

Ginsenoside Rg1 treatment ameliorated sepsis-induced AKI by reducing iron deposition and lipid peroxidation and elevating the expression of GPX4 and FSP1