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As not all patients with FEP will be able to increase GSH in accordance with glutamate levels, we expected a reduction in the strength of correlation between the GSH and glutamate levels in patients compared with healthy controls (hypothesis 2)

However, it was found that the presence of polymorphisms in the genes encoding this enzyme can contribute to an increased risk of CRC development in the case of interaction with other factors, such as diet or smoking [33]

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To further explore the role of GPx1 in glucose metabolism in PDA cells upon glucose deprivation, key signature enzymes in the glycolysis cascade were examined

Improvements in subscales of the Vineland Adaptive Behavior Scale, the Aberrant Behavior Checklist, the Autism Symptom Questionnaire and the Behavioral Assessment System for Children were significantly greater in the folinic acid group as compared with the placebo group