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The lack of a well-defined primary receptor for DSIP has hampered mechanistic research if such a receptor exists, it has not been convincingly identified and cloned, leading some researchers to propose that DSIP may work through non-receptor-mediated mechanisms or through promiscuous interactions with multiple receptor systems

In another human clinical trial conducted on a sample of pulmonary tuberculosis patients, epitalon did not appear to correct pre-existing structural aberrations of chromosomes associated with telomere degradation, but did appear to exert a protective effect against the future development of additional chromosomal aberrations

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