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Description

Nature 320 , 584588 (1986)

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These effects can be explained by the activation of strong defense responses in these mutants, including the suberinization of infection thread walls in sym33-3 nodules ( sym40-1 nodules (Tsyganova et al., 2009)

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TP53, tumor protein 53

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Conclusions Our data indicates that GSH participates in retarding apoptosis in antihormone-resistant human breast cancer cells and that depletion of this molecule by BSO may be critical in predisposing resistant cells to E 2 -induced apoptotic cell death

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Tan JS, Wang JJ, Flood V, Smith W, Mitchell P

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