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data showed in this study enhanced cell proliferation of HSC treated with BSA-AGEs (CML) and increased production of -SMA

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The question remains: To what extent can any one test or biomarker adequately assay all of the individual pathways involved in such pathways and how can that information be best used to guide individual risk or even treatment

Abstract Prior studies have shown that inhibiting ferroptosis could facilitate neural repair following traumatic spinal cord injury (TSCI), yet the underlying mechanisms remain unclear