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These results suggest that Cu is a risk factor for pulmonary fibrosis that activates the ROS-autophagy pathway and that flavonoids can reverse this process ( in vitro and in vivo evidence of a causal relationship between Cu exposure and pulmonary fibrosis and suggest that EMT changes are a key mechanistic event in Cu-induced pulmonary fibrosis

Systemic Inflammation but not Oxidative Stress Is Associated with Physical Performance in Moderate Chronic Obstructive Pulmonary Disease