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Description

Specifically, the endoplasmic reticulum enhances autophagy through the IRE1/JNK signaling pathway, leading to the degradation of FTH1

Furthermore, the internal space and unique cavity of the cyclodextrin-based metalorganic framework can increase bioactive molecule instability and give the possibility of prolonged release of the loaded bioactive component

NF kappa B and AP-1 mediate transcriptional responses to oxidative stress in skeletal muscle cells

Primary Handling Editors: Jin-Min Nam and Tobias Goris

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