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Research shows that stress-triggered neuroinflammation increases TLR4 receptor expression, indicating a mechanistic pathway by which stress-induced inflammation may facilitate depressive behavior [57]

Release of binding proteins and stored B12 occurs when hepatocytes are damaged

Emerging studies even suggest that erythrocytes exhibit neuron-like properties: they can respond to and metabolize circulating neurotransmitters, carry out transamination reactions, and release microparticles enriched with bioactive lipids, metabolites, and glycolytic enzymes

doi: 10.1007/s11033-019-05136-4

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