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At the same time, the expression levels of pro-caspase-3 and IB-a were significantly upregulated upon pretreatment of human colon cells with NF-B activity-inhibiting drugs, suggesting that inhibition of NF-B can reduce the apoptosis of intestinal epithelial cells (Cao et al., 2020)

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Additionally, mitochondrial defects contribute to increased ROS levels and decreased ATP production, which is vital for maintaining cellular homeostasis, particularly in energy demanding cells like RGCs (127)

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