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Microscopically, solid granules in a chronic inflammatory cell infiltrate present the same histopathologic aspect in both graphite and amalgam tattoos

If this is true, then it is likely that either the upregulated process of melanin biosynthesis, the increased amount of melanin in the melanocytes, or both, occurring in the fields of benign oral melanin hyperpigmentation, constitute risk factors for transformation into oral mucosal melanomata

This is significantly longer than the few seconds of endogenous -MSH, but remains short compared to long-acting analogs such as subcutaneous afamelanotide implant (release over 60 days)

Additionally, ultrasound vocalization (USV) testing was used to measure communication deficits

Physiological gingival melanin hyperpigmentation is often more pronounced in the anterior than in the posterior part of the mouth, and the buccal/labial surfaces are more intensely pigmented than the lingual/palatal [35]