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Substrate binding of c-Jun induces dimeric DDD-Ube2e2-COP1 formation Since we hypothesized that the purified stacked DDD-Ube2e2-COP1 protein assemblies might represent inactive states, we next sought to investigate how c-Jun is recognized by the CULLIN4 DET1-COP1 E3 ligase

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This mitigates the adverse effects (AEs) associated with long courses of high-dose corticosteroids to some extent

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Methods: Liver injury and liver fibrosis were induced by carbon tetrachloride(CCl 4 ) in wild-type mice and Ccr2 knockout ( Ccr2 -/- ) mice