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Description

It reduces H 2 O 2 -induced GSSG production, IL-1 levels, and cell proliferation in TAO orbital fibroblasts (142)

Indeed, the treatment significantly induced the phosphorylation of the insulin-like growth factor-I (IGF-I)-mediated phosphatidylinositol 3 kinase/serinethreonine kinase (PI3K/Akt) pathway [78], modulated apoptosis by suppressing Bad (a proapoptotic protein), upregulating the pro-survival proteins p-Bad and Bcl-xL, and increasing the expression of the proliferating cell nuclear antigen (PCNA)

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