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Mousa, King Abdulaziz University, Saudi Arabia Reviewed by Taras P

E-cadhedrin contributes to tumor progression by increasing proliferation, invasion, and metastasis [19]

The effective binding and functional modulation of the targets is enabled by advanced protein engineering methodologies, including rational design, chemical protein synthesis, and mirror-image display, which allow precise control over the specificity, stability, and pharmacological properties

This exhausted state renders T cells dysfunctional and reversing or preventing this phenotype is a major goal in cancer immunotherapy ( IFNG and GZMB ) and a gain of ChARs at exhaustion gene loci ( PDCD1 , HACVR2, and BATF ) compared with non-exhausted T cells ( thymocyte selection-associated HMG BOX ( TOX ) locus ( Figure 2 )