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Much of this evidence comes from cell and animal studies, not large human trials

10.1089/ars.2010.3600 82 LiB.PrattD

Supplementation studies which have utilized exogenous forms of glutathione are not that common due to the poor ability of glutathione to stay intact throughout the cell with most of it being found to be degraded in the extracellular compartment[18]

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Taken together, these data argue that LCN2 represents an important, tumor-exploited iron trafficking axis that complements canonical TFR1/DMT1 routes and should be considered when assessing tumor iron biology and designing iron-targeted interventions