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Increasing p16INK4a expression decreases forebrain progenitors and neurogenesis during ageing

50,51 This energy deficiency triggers the collapse of the mitochondrial membrane potential ( m ) and the release of proapoptotic factors, which activate the caspase cascade and induce apoptosis or senescence

Other glutamate receptors, including kainate receptors (KARs) and metabotropic glutamate receptors, can control long-term and short-term synaptic plasticity [76, 77]

Finally, pharmacological research is needed to ensure that drugs are delivered to the necessary organs, such as the inflammatory joints of RA, with optimum safety and efficiency in a wide range of autoimmune diseases

doi: 10.1016/j.bcp.2007.07.007