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Collectively, these advances demonstrate PBNs unique capacity for precision hemostatic modulation across traumatic, neurovascular, and immune-mediated bleeding disorders through biomimetic coagulation potentiation, ROS-mediated neuroprotection, and pathological antibody neutralization [128, 171]

9 Notably, insulin signalingparticularly through the PI3K-Akt pathwayplays a dual role: it not only regulates glucose metabolism but also promotes SC differentiation and remyelination, thereby mitigating DPN progression

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