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the conversion of methylmalonic acid to succinyl coenzyme A

However, recent work also indicates that GO can directly damage cells via interactions with various biomacromolecules 18,19,20,21,22,23,24

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When JNK is activated in an inflammatory state, it further induces the phosphorylation of the linker region of Smad3 (pSmad3L), thereby targeting downstream plasminogen activator inhibitor-1 (PAI-1) activation and participating in fibrosis (167)