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As stated above, according to several evidence, acute COVID-19 damage to the CNS may play a central role in the pathogenesis of neuropsychiatric symptoms through several pathophysiological mechanisms: dysregulation of inflammation, astrocytic glial damage, glutamatergic impairment (shift in tryptophan metabolism towards kynurenine pathway, Nf-B, oxidative stress, glutamatergic and excitotoxic damage) and possible glymphatic impairment [15, 46, 82]

Glutathione (-glutamylcysteinylglycine) is a ubiquitous tripeptide thiol which plays a crucial role in oxidative stress defence mechanism of the cell

When stomach acid flows back into the esophagus, it can cause burning pain that is often worse after lying down all night

Venous thrombosis has been reported to be more frequent in vitamin B 12 deficient subjects than in controls

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