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Ischemia-reperfusion injury (IRI) occurred inevitably at the early phase after liver transplantation (LT) spawns a significant risk of HCC recurrence

Loss of this feedback enhances IRS1 activity, leading to increased PI3K signaling and subsequent AKT phosphorylation

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Research from Johns Hopkins Medicine on the gut-brain axis has established bidirectional communication between the gut and brain via the vagus nerve and through neurotransmitter production