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Mechanistically, the metabolic benefits of Godex depend on enhanced activity of CrAT, which modulates mitochondrial acetyl-CoA metabolism and thereby improves insulin sensitivity and hepatic lipid homeostasis (80), whereas some human studies report only modest reductions in liver enzymes without significant changes in broader metabolic parameters (64)

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This will also result in a competition for citrate synthase by both propionyl-CoA and acetyl-CoA, further blocking the metabolism of metabolic pathways that produce acetyl-CoA as an end product, such as the fatty-acid oxidation pathway

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