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As discussed previously, most studies regarding tumor amino acid metabolism focus on the regulatory mechanism, which contributes to tumor growth (as an essential nutrition, energy source, or raw materials), epigenetic modification, tumor immunity, and ferroptosis

also reported that Paclitaxel exposure induces ROS production, as H 2 O 2 accumulation showed to be an early and crucial step in paclitaxel cytotoxicity [32]

BPC-157 exhibits cytoprotective, anti-inflammatory, and angiogenic properties across various tissues

The results indicated that MT significantly reduced the inflammatory response in the burn mouse model, as evidenced by a decrease in the secretion of pro-inflammatory cytokines (TNF-, IL-1, and IL-6) and a significant increase in the secretion of the anti-inflammatory cytokine IL-10