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People whose injections have been stopped/delayed may want to think about drawing GPs attention to the potential for permanent neurological damage if a person is under treated

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Pharmacokinetic studies revealed [6] : Orally administered AOD9604 was well absorbed in pigs with rapid degradation kinetics Primary degradation products were peptide fragments missing 2-3 amino acids from termini Both degradation products retained some reduced anti-lipogenic activity Rat whole-body radiography showed similar organ distribution patterns following both intravenous and oral administration This distribution pattern supports systemic absorption via the oral route in laboratory models

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There is research suggesting that methylcobalamin may be particularly supportive in neurological conditions compared to cyanocobalamin, likely because of its direct involvement in methylation and nerve tissue repair