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The hypothetical framework was that excessive glutamate neurotransmission in early psychosis underpins the emergence of positive symptoms and results in loss of presynaptic glutamate terminals and the development of persistent negative symptoms in established illness

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[4] Greenhaff PL, et al, 2006: Insulin stimulates L-carnitine accumulation in human skeletal muscle

As a result, the production of mature IL-1 was decreased following activation of NLRP3 by nigericin or ATP, with no effect on pyroptosis